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The upregulation of Trx1, however, did not seem to compensate fully for the loss of TRP14 activities, because when assessing cystine reduction using BODIPY-labeled cystine, which becomes fluorescent upon reduction of its disulfide (Pader et al, 2014), TRP14 deletion severely hampered the strong fluorescence seen in control cells (Fig

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KAT8-catalyzed lactylation promotes eEF1A2-mediated protein synthesis and colorectal carcinogenesis

Notoginsenoside Ft1 promotes fibroblast proliferation via PI3K/Akt/mTOR signaling pathway and benefits wound healing in genetically diabetic mice